From: Persistent CT nephrograms following cardiac catheterisation and intervention: initial observations
Publication | Observations | Change in renal function | Contrast media |
---|---|---|---|
Older et al. (1976) [13] | Persistent nephrograms on plain films 24 h after angiography in 17/90 (19%) of patients of which 9/17 (53%) did have and 8/17 (47%) did not have significant change in renal function | ↑SCr ≥20% or 26.40 μmol/l | HOCM |
Love et al. (1989) [14] | Delayed CT cortical enhancement of 141.6 HU 22–26 h after angiography in 1/50 (2%) with any significant change in renal function. Cortical enhancement of 55–110 HU classified as “subclinical renal impairment” | ↑SCr = 150% of baseline or 88 μmol/l | HOCM and LOCM |
Jakobsen et al. (1992) [24] | Cortical attenuation of 52 ± 6 HU at 8–32 h in 40 healthy male volunteers after CECT. No change in renal function. (No focal CT nephrograms reported) | ↑SCr, Cr Cl and urinary biomarkers | LOCM, LOCM ionic dimer |
Yamazaki et al. (2001) [11] | Delayed CT cortical enhancement 16–21 h after TACE in 81/180 (45%) and nephropathy in 11/180 (6%) of treatments. Minimal cortical retention >50 HU and severe retention >100 HU | ↑SCr ≥44 μmol/l or ≥25% | HOCM, LOCM, LOCM ionic dimer |
Monsky et al. (2009) [12] | CT renal nephrograms 24 h post TACE in 14/60 (23.3%) treatments. Global nephrograms associated with significant (p = 0.031) change in SCr at 24 h. (Delayed segmental nephrograms associated with procedural factors) | ↑SCr at 24 h and 24 h from baseline pre-TACE values | IOCM and LOCM |