Acute hepatocellular injury
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Hepatosplenomegaly, gallbladder wall thickening, reduced and heterogeneous liver enhancement, abdominal ascites, reduced portal flow, periportal edema
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Chronic hepatocellular injury
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Capsular retraction, confluent fibrosis, pseudocirrhosis (i.e., liver surface nodularity, multifocal capsular retraction, decreased liver size, enlargement of the caudate lobe, signs of portal hypertension)
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Nonalcoholic fatty liver disease
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Hepatic steatosis (lower attenuation of the hepatic parenchyma on CT, signal drop on opposed phase compared to the in-phase images on MRI)
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Sinusoidal obstruction syndrome
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Diffuse form: patchy liver enhancement with a mosaic appearance, mainly in the periphery of the right lobe; focal form: lack of rim-enhancement in the arterial and portal venous phases, lack of diffusion restriction on DWI, intermingled hypointensity and ill-defined margins in the hepatobiliary phase
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FNH-like lesions
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Usually multiple; homogenous arterial contrast enhancement and isoattenuation to the surrounding liver parenchyma in the portal venous and delayed phase; on hepatobiliary phase MRI iso or hyperintense or with a ring or doughnut-like appearance (i.e., hyperintense periphery and central hypointensity)
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