|Brain||Lead exposure||MRI||Decreased NAA/Cr ratios in grey matter wich suggest neuronal loss.|
Finding were more remarkable in frontal regions .
|Brain||DTI||DTI shows abnormalities in radial diffusivity which means changes in myelin sheath thickness and organisational characteristics.|
|Brain||Cerebral stroke||Carotid ultrasonography||Increased carotid intima-media thickness, decreased carotid arteries distensibility, early atherosclerotic lesions .|
|Brain||Pesticide neurotoxicity||MRS||Regional elongation in the cerebral surface with changes in the gyrus rectus, superior frontal gyrus, cuneus and precuneus along the mesial wall of the right hemisphere.|
Anatomical alterations in the mesial and dorsal surfaces of the left superior frontal gyrus .
|Thorax–lungs||Smoke inhalation||Radiography||Chest radiography shows three stages:|
1. The acute stage < 24 h after exposition: normal characteristics of the lungs.
2. The subacute stage 2–5 days after exposition: manifests as pulmonary oedema, atelectasis, pulmonary micro-embolism, and adult respiratory distress syndrome (ARDS).
3. The delayed stage > 5 days after exposition: pneumonia and pulmonary thromboembolism .
|Lungs||Organophosphates poisoning||Radiography||Lung oedema that could appear within 24 hours after exposition .|
|GI||Chronic inflammatory bowel disease||Aortic ultrasound||Increased aortic intima-media thickness .|
|Heart||Myocardial infarction||Carotid ultrasound||Increased carotid intima-media thickness .|
|Arteries||Artery hypertension||Aortic and brachial ultrasound||Higher stiffness and lower elasticity .|